![]() ![]() If you are a member of an institution with an active account, you may be able to access content in one of the following ways: Get help with access Institutional accessĪccess to content on Oxford Academic is often provided through institutional subscriptions and purchases. Whereas pharmacological treatment is often only moderately effective, catheter ablation of the accessory pathway is highly effective with a low complication rate. Due to the often incessant nature of PJRT, patients may be at risk for tachycardia-induced cardiomyopathy. Atypical atrioventricular nodal reentry tachycardia and focal atrial tachycardia are important differential diagnoses. The 12-lead electrocardiogram during PJRT shows negative P waves in the inferior lead II, III, and aVF, with a long RP interval. PJRT is a rare form of supraventricular tachycardia and can be found in all age groups but the majority of affected patients are children and young adults. The accessory pathway is most commonly located in the posteroseptal region however, other locations have been described. ![]() Can cause cardiomyopathy.The term permanent junctional reciprocating tachycardia (PJRT) describes an orthodromic atrioventricular reentry tachycardia using a usually concealed slowly conducting accessory pathway with decremental properties as the retrograde limb. May not respond to DC cardioversion or adenosine. Neonates (then not again until adulthood) Saw tooth flutter waves (often only appreciated with AV block from adenosine)įixed but may appear irregular depending on AV conduction Up to 500 beats/min in neonates (300 beats/min in children) with variable AV conduction being common Neonates and infants (220 (commonly 250-300 in infants) If unsuccessful, or child deteriorates, proceed to medical treatment as per the algorithmĭO NOT perform Eyeball pressure or carotid sinus massage as they are no longer recommended.Continuous cardiac monitoring should be applied prior to performing.Vagal manoeuvres are safe, minimally invasive and effective for reverting SVT in a hemodynamically stable child or infant.*Adapted from the Sydney Children’s Hospital Network Guideline on SVT 1. Blood tests provide no added benefit to the diagnosis or management and therefore are not recommended, unless considering other causes of SVT.CXR can be considered if clinical signs of heart failure present.(See additional notes below for further details) Are P-waves present and what is their morphology?.Is there a broad or narrow QRS complex?.Red Flags Unwell appearance, hypotension, drowsiness/altered conscious state Note: BP machines and pulse oximeters may be unreliable at common SVT ratesġ2 lead ECG (pre and post reversion techniques) Typically present late, with signs of congestive heart failure (respiratory distress, hepatomegaly, oedema) Irritability, poor feeding, lethargy and sweatingĬhild complains of feeling ill, breathless, chest pain, nausea, dizziness or sweating Tolerate tachycardia well, making early signs and symptoms initially subtleĪble to perceive palpitations and therefore present earlier with less clinical signs or symptoms The priority is to identify the child in shock (pallor, poor perfusion, decreased consciousness, hypotension) and proceed immediately to resuscitation (see flowchart below) Factors that may contribute to tachycardia (eg sepsis, pain, dehydration, anxiety, and fever) should also be considered and addressed when managing a patient presenting with a tachyarrhythmia.Always assume a broad complex tachycardia is due to VT rather than SVT with aberrancy, unless there is clear evidence it is not VT.Onset and offset are abrupt, and p-waves are either not visible or seen after the QRS complexes (See additional notes below for important features when differentiating between different causes of narrow complex tachyarrhythmias) SVT typically has a fixed rate, usually >220 bpm.Other causes: include sinus tachycardia, atrial flutter, ectopic atrial tachycardia and junctional ectopic tachycardia. ![]() Adolescents: more commonly caused by atrioventricular nodal re-entry (AVNRT).Younger children: usually caused by atrioventricular re-entry (AVRT), including Wolff-Parkinson White syndrome.SVT is one cause of narrow complex tachycardia.SVT is an abnormally fast heart rate originating from above the ventricles.Vagal manoeuvres should only be attempted in a child who is clinically stable.Greater than a third of new onset SVT occurs in the first few weeks of life, commonly presenting after many hours with signs of heart failure.Most SVT in children is due to a re-entrant mechanism and usually occurs in otherwise normally well children Supraventricular tachycardia (SVT) is common in infancy and childhood.Resuscitation: Care of the seriously unwell child Recognition of the seriously unwell neonate and young infant Cardiac telemetry Key points ![]()
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